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Alterations to glyma.Zhuang_Xue_2021.yml on 17 December 2024
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ScottKalberer committed Dec 18, 2024
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Showing 1 changed file with 27 additions and 18 deletions.
45 changes: 27 additions & 18 deletions Glycine/max/studies/glyma.Zhuang_Xue_2021.yml
Original file line number Diff line number Diff line change
Expand Up @@ -3,17 +3,23 @@ scientific_name: Glycine max
gene_symbols:
- GmSPX5
gene_symbol_long: SYG1/Pho81/Xpr1 5
gene_model_pub_name: Glyma10g40820
gene_model_full_id: glyma.Wm82.gnm1.ann1.Glyma10g40820
gene_model_pub_name: Glyma10G40820
gene_model_full_id: glyma.Wm82.gnm4.ann1.Glyma.10G261900
confidence: 5
curators:
- Marlene Dorneich-Hayes
- Scott Kalberer
comments:
- SPX5 is most strongly expressed in nodules.
- Phosphate deficiency promotes expression of SPX5 in nodules but decreases SPX5 expression in roots. Nitrogen deficiency decreased SPX5 expression in roots.
- SPX5 is upregulated by arbuscular mycorrhizal fungal infection in phosphate-deficient conditions.
- SPX5 promotes binding of NF-YC4 to the promoter of ASL6, increasing expression of ASL6 during nodule development.
phenotype_synopsis: SPX5 upregulates ASL genes to positively regulate nodule number. Unlike other SPX genes, SPX5 does not affect expression of PHR genes.
- GmSPX5 is most strongly expressed in root nodules.
- Phosphate deficiency promotes expression of SPX5 in nodules but decreases
SPX5 expression in roots. Nitrogen deficiency decreased SPX5 expression in
roots.
- SPX5 is upregulated by arbuscular mycorrhizal fungal infection in
phosphate-deficient conditions.
- SPX5 promotes binding of GmNF-YC4 to the promoter of GmASL6, increasing
expression of ASL6 during nodule development.
phenotype_synopsis: GmSPX5 upregulates GmASL genes to positively regulate nodule
number. Unlike other SPX genes, SPX5 does not affect expression of PHR genes.
traits:
- entity_name: regulation of biological process involved in symbiotic interaction
entity: GO:0043903
Expand All @@ -22,29 +28,32 @@ traits:
- relation_name: positively regulates
relation: RO:0002213
references:
- citation: Zhuang, Xue et. al., 2021
- citation: Zhuang, Xue et al., 2021
doi: 10.1111/tpj.15520
pmid: 34587329
- citation: Yao, Tian et. al., 2014
- citation: Yao, Tian et al., 2014
doi: 10.1093/aob/mcu147
pmid: 25074550
- citation: Yang, Lan et. al., 2022
- citation: Yang, Lan et al., 2022
doi: 10.1111/jipb.13207
pmid: 34962095
---
scientific_name: Glycine max
gene_symbols:
- GmNF-YC4
gene_symbol_long: Nuclear Factor Y subunit C4
gene_model_pub_name: Glyma06g17780
gene_model_full_id: glyma.Wm82.gnm1.ann1.Glyma06g17780
gene_symbol_long: Nuclear Factor Y Subunit C4
gene_model_pub_name: Glyma06G17780
gene_model_full_id: glyma.Wm82.gnm4.ann1.Glyma.06G169600
confidence: 4
curators:
- Marlene Dorneich-Hayes
- Scott Kalberer
comments:
- NF-YC4 positively regulates nodule number and nitrogenase activity.
- SPX5 promotes binding of NF-YC4 to the promoter of ASL6, increasing expression of ASL6 during nodule development.
phenotype_synopsis: NF-YC4 is a transcription factor which upregulates ASL genes to positively regulate nodule number.
- GmNF-YC4 positively regulates nodule number and nitrogenase activity.
- GmSPX5 promotes binding of NF-YC4 to the promoter of GmASL6, increasing
expression of ASL6 during nodule development.
phenotype_synopsis: GmNF-YC4 is a transcription factor which upregulates GmASL
genes to positively regulate nodule number.
traits:
- entity_name: DNA-binding transcription factor activity
entity: GO:0003700
Expand All @@ -55,9 +64,9 @@ traits:
- relation_name: positively regulates
relation: RO:0002213
references:
- citation: Zhuang, Xue et. al., 2021
- citation: Zhuang, Xue et al., 2021
doi: 10.1111/tpj.15520
pmid: 34587329
- citation: Yang, Lan et. al., 2022
- citation: Yang, Lan et al., 2022
doi: 10.1111/jipb.13207
pmid: 34962095

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