From 1a42066848273712a8da80576328ee1ec299c4a0 Mon Sep 17 00:00:00 2001 From: StevenCannon-USDA Date: Fri, 1 Dec 2023 11:08:14 -0600 Subject: [PATCH] Remove some mis-placed files --- phavu_Davila-Delgado_Flores-Canul_2023 | 27 -------------------------- vigra_Liu_Zhang_2022 | 23 ---------------------- 2 files changed, 50 deletions(-) delete mode 100644 phavu_Davila-Delgado_Flores-Canul_2023 delete mode 100644 vigra_Liu_Zhang_2022 diff --git a/phavu_Davila-Delgado_Flores-Canul_2023 b/phavu_Davila-Delgado_Flores-Canul_2023 deleted file mode 100644 index 35f1001..0000000 --- a/phavu_Davila-Delgado_Flores-Canul_2023 +++ /dev/null @@ -1,27 +0,0 @@ -gene_symbols: - - SYMRK (in Phaseolus vulgaris, Lotus japonicus, and Arachis hypogaea) - - DMI2 (in Medicago truncatula) - - NORK (in Medicago sativa) -gene_symbol_long: SYMbiosis Receptor-like Kinase -gene_model_pub_name: Phvul.002G143400 -gene_model_full_id: phavu.G19833.gnm1.ann1.Phvul.002G143400 -confidence: 5 -curators: - - Marlene Dorneich-Hayes -comments: - - SYMRK is activated in root hairs by phosphorylation in response to the perception of NOD factors - - SYMRK-EGFP (enhanced green fluorescence protein) is endocytosed from the plasma membrane to intracellular puncta and affects the SYMRK signaling pathway - - SYMRK-suppressed mutants can't form infection threads and therefore can't form functional nodules, though they do form pseudo-nodules lacking symbiotic bacteria and fungi - - SYMRK overexpressing mutants form non-functional nodules without rhizobia -phenotype_synopsis: SYMRK is necessary for nodule organogenesis. -traits: - - entity_name: bioloical process involved in symbiotic interaction - entity: GO:0044403 - - entity_name: kinase activity - entity: GO:0016301 - - entity_name: positive regulation of receptor-mediated endocytosis - entity: GO:0048260 -references: - - citation: Dávila-Delgado, Flores-Canúl, et al., 2023 - doi: 10.1007/s00425-023-04116-0 - pmid: 36928335 diff --git a/vigra_Liu_Zhang_2022 b/vigra_Liu_Zhang_2022 deleted file mode 100644 index bd35c82..0000000 --- a/vigra_Liu_Zhang_2022 +++ /dev/null @@ -1,23 +0,0 @@ -gene_symbols: - - CCA1L26 -gene_symbol_long: Circadian Clock Assoicated 1 Like 26 -gene_model_pub_name: Vradi05g00310 -gene_model_full_id: vigra.VC1973A.gnm6.ann1.Vradi05g00310 -confidence: 3 -curators: - - Marlene Dorneich-Hayes -comments: - - VrCCA1L26 shares homology with AtCCA1, AtLHY, and GmLCL genes. - - In transgenic Arabidopsis, VrCCA1L26 delays flowering time by supressing Constans (CO), Flowering locus T (FT), Supressor of Overexpression of Constans 1 (SOC1), and Timing of Cab Expression 1 (TOC1). - - VrCCA1L26 expression is affected by photoperiod and is supressed by expression of TOC1. - - VrCCA1L26 may play a role in isoflavonoid synthesis, stress response, hypocotyl growth, seed germination, and leaf senescence. This has not been experimentally validated. -phenotype_synopsis: VrCCA1L26 delays flowering time in transgenic Arabidopsis thaliana, but its function in Vigna radiata has not been experimentally validated. -traits: - - entity_name: transcription factor binding - entity: GO:0008134 - - entity_name: photoperiod-sensitive flowering time trait - entity: TO:0000934 -references: - - citation: Liu, Zhang, et. al., 2022 - doi: 10.1186/s12864-022-08620-7 - pmid: 35581536